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Welcome to the POZ/AIDSmeds Community Forums, a round-the-clock discussion area for people with HIV/AIDS, their friends/family/caregivers, and others concerned about HIV/AIDS.  Click on the links below to browse our various forums; scroll down for a glance at the most recent posts; or join in the conversation yourself by registering on the left side of this page.

Privacy Warning:  Please realize that these forums are open to all, and are fully searchable via Google and other search engines. If you are HIV positive and disclose this in our forums, then it is almost the same thing as telling the whole world (or at least the World Wide Web). If this concerns you, then do not use a username or avatar that are self-identifying in any way. We do not allow the deletion of anything you post in these forums, so think before you post.

  • The information shared in these forums, by moderators and members, is designed to complement, not replace, the relationship between an individual and his/her own physician.

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Recent Posts

Pages: 1 ... 7 8 [9] 10
Am I Infected? / Please help me
« Last post by ozguy on Yesterday at 07:39:26 AM »
Hi, I engaged in oral sex last tuesday. I was the giver. It was very brief for only 1 or 2 minutes. After that, I realised my lips had wound and was bleeding. I am terrified right now. I have no idea my partner HIV status. I would like to ask few questions:
1. Can HIV be transmitted through saliva as I kissed, but then it was also only for 2-3 minutes
2. What is the chance of me catching HIV if he is HIV positive?
3. How long can HIV survive in the air? As he probably had ejaculated and this activity done in a steam room, will hot air make the virus survive longer in the air?

Am I Infected? / Re: Blood during sex
« Last post by Jeff G on Yesterday at 05:52:58 AM »
Sexual fluids are not infectious once outside the body so what you describe about putting the condom back on did not increase your risk. At the end of the day testing is the only way to know your HIV status . 

Hiv is a fragile, difficult to transmit virus that is primarily transmitted INSIDE the human body, as in UNPROTECTED anal or vaginal intercourse where the virus never leaves the confines of the two bodies. Once hiv finds itself outside the body, small changes in temperature, and pH and moisture levels all quickly damage the virus and render it unable to infect.

Am I Infected? / Re: Blood during sex
« Last post by fs1epop on Yesterday at 05:44:26 AM »
Sorry Andy

One thing I forgot to mention is that I originally entered him with the condom then lost my erection.  I pulled out, took the condom off, got hard again, then put it back on and went back in.  That's why it broke I think.

I'm also concerned that I put the condom on inside out the second time around.  Could this be a risk (ie having pick up some anal mucus)?

Am I Infected? / Re: Blood during sex
« Last post by Jeff G on Yesterday at 05:42:11 AM »

I have heard you and others on this forum say that you have never seen the top infected after a condom break.  Is this during heterosexual vaginal sex or does it include homosexual anal sex?

When we say condom breaks and brief insertions rarely lead to HIV infections for the insertive partner we mean anal or vaginal . Just test at the appropriate time and you will will be OK .
Am I Infected? / Re: Blood during sex
« Last post by fs1epop on Yesterday at 05:33:28 AM »
Thanks Andy

Yes you are right I was the top in this incident, and as I say I was probably only inside him for about 5-10 seconds in total including the break.

We had a talk afterwards and he told me he was negative, tested 6 weeks ago, but as you say who knows.  I would take his status as unknown. 

I have heard you and others on this forum say that you have never seen the top infected after a condom break.  Is this during heterosexual vaginal sex or does it include homosexual anal sex?


Research News & Studies / Re: Two, Once-Daily HIV Meds Approved On Same Day
« Last post by Jeff G on Yesterday at 05:02:13 AM »
I like the second one ... you could say I'm on the Taz man the Taz saved my life .
Living With HIV / Re: myelitis anyone
« Last post by guitargal on Yesterday at 03:41:45 AM »
wow read some of your story here and way to go!
so many tough roads we go down and then a bright plateau..

yes your info helps people..

inspired me to get off my ass and try to get better…
now i need to get some stretching and exercise in..daily..even if it is a little at a time..
thanks for your onsite and sharing.
Forums Gatherings / Re: Official choice of place and time for AMG 2015
« Last post by guitargal on Yesterday at 03:00:29 AM »
This sounds like fun! Can I come?
Clean and Sober 22+ years so unfortunately weed and Budweiser not choices 😝😝😝😝😝 LOL BUT I KEEP UP WITH MY LEGAL MEDS! My job AHS been a little less stressful because I found the line for don't give a fuck and giving great customer service! And my supervisor is useless!
Research News & Studies / Re: Latency found to be in quiescent T cells
« Last post by tryingtostay on Yesterday at 12:20:26 AM »
I think this is the same story but in an expanded version:


Drugs for HIV have become adept at suppressing infection, but they still can't eliminate it. That's because the medication in these pills doesn't touch the virus' hidden reserves, which lie dormant within infected white blood cells. Unlock the secrets of this pool of latent virus, scientists believe, and it may become possible to cure - not just control - HIV.

In a study published Thursday (January 29) in Cell, researchers lead by Zanvil A. Cohn and Ralph M. Steinman Professor Michel C. Nussenzweig at Rockefeller University and their collaborators describe new insights on which cells likely do, and do not, harbor this lurking threat.

"It has recently been shown that infected white blood cells can proliferate over time, producing many clones, all containing HIV's genetic code. However, we found that these clones do not appear to harbor the latent reservoir of virus," says study author Lillian Cohn a graduate student in Nussenzweig's Laboratory of Molecular Immunology. "Instead our analysis points to cells that have never divided as the source of the latent reservoir."

HIV belongs to a family of viruses that insert themselves directly into the host cell's genome where they can hide out quietly after the initial infection. HIV mostly targets CD4 T lymphocytes, a type of T cell involved in initiating an immune response.

When HIV integrates itself into the genetic code of a CD4 T cell, it may produce an active infection, hijacking the cell to produce more copies of itself in order infect other cells, and killing it in the process. Antiretroviral drugs that suppress HIV infection work by disrupting this hijacking. But the virus may also fail to produce an active infection, remaining a quiet, tiny fragment of DNA tucked within the host cell's genome. If so, the drugs have nothing to disrupt, and the infection remains latent.

Most often, however, what happens is actually something in between. While the virus does manage to get at least some of itself into the T cell's genome, problems with the process leave it incapable of hijacking the cell to replicate itself. But those few successful integrations still do damage, and the resulting depletion in the victim's immune system leaves him or her vulnerable to potentially fatal opportunistic infections years, or even decades, after the initial infection.

"If a patient stops taking antiretrovirals, the infection rebounds. It is truly amazing that the virus can give rise to AIDS 20 years after the initial infection," Cohn says.

Researchers think the reservoir of latent virus may be hiding out in a type of CD4 T cell: long-lived memory cells that help the immune system remember particular pathogens. When these cells encounter a pathogen they have previously seen, they spur the proliferation of T cells tuned to recognize it, in a process called clonal expansion. Prior research has suggested clonal expansion is crucial to maintaining HIV's latent reservoir.

Following up on work initiated by Mila Jankovic, a senior research associate in the lab, Cohn and her colleagues examined cloned and unique CD4 T cells in blood samples from 13 people infected with HIV. An analytical computational technique developed by Israel Tojal da Silva, a research associate in the lab, made it possible to identify integration sites into which HIV had inserted itself within individual cells.

"Given the size of the human genome, it is highly unlikely the virus would insert itself in exactly the same place more than once. So, if multiple cells contained virus with identical integration sites, we classified them as clones. Meanwhile if a cell had a unique integration site, one not shared with any other cell, then we assumed that cell was unique" Cohn says.

The researchers tested 75 viral sequences taken from the expanded clones of cells to see if they had the potential to produce more of the virus. None could.

"While we cannot rule out the possibility that a rare clone of cells may contain an active virus, it appears most likely that latent reservoir - and the potential target for therapies meant to cure HIV - resides in the more rare single cells containing unique integrations," Cohn says.
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